“Study links primary insomnia to a neurochemical abnormality in young and middle-aged adults. The study is the first to show a specific neurochemical difference in the brains of adults with primary insomnia.”
At last! I knew it, I said it in INSOMNIAC: insomnia is not (necessarily) about psychopathology, it’s about neurophysiology I always knew there was some neurobiological/neurochemical difference between me and people whose sleep comes down like a black-out curtain, sweeps them under like a wave. Here’s what I wrote:
“What if, in the not too distant future, people are saying, ‘we used to think insomnia was [about habits, attitudes, psychopathology]… but we now know there’s too little of a crucial neurotransmitter, some substance that’s not being produced… some glitch in the brain’s extraordinary circuitry that leaves insomniacs without the neurological wherewithal to transition to or maintain normal sleep’?”
I knew the research would bear this out. I didn’t know how soon.
John Winkelman, lead researcher, says this “may increase the legitimacy of those who have insomnia.” Yes, it will! Very exciting research! Go, Winkelman!
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